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In studies of other receptor tyrosine kinases implicated while in the oncogenesis of GIST, nilotinib realized strong and selective inhibition of PDGFRα and PDGFRβ. As is the situation with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells transformed by PDGFRAesomeprazole will lessen the extent or influence of nilotinib